Josephson's Clinical Cardiac Electrophysiology
Produs indisponibil momentan. Pentru comenzi va rugam trimiteti mail la adresa depozit2@prior.ro sau contactati-ne la numarul de telefon 021 210 89 28 Vedeti mai jos alte produse similare disponibile.
Disponibilitate: Acest produs nu este momentan in stoc
Autor: Mark E. Josephson MD
Editura: LWW
Limba: Engleza
Nr. pagini: 890
Coperta: Hardback
Dimensiuni: 1.2 x 9 x 11 inches
An aparitie: October 2, 2015
Description:
Turn to this updated, classic text for a thorough understanding of the mechanisms of cardiac arrhythmias and the therapeutic interventions used to treat them. Josephson's Clinical Cardiac Electrophysiology, 5th Edition delivers Dr. Mark Josephson's unparalleled guidance on the electrophysiologic methodology required to define the mechanism and site of origin of arrhythmias – enabling you to choose the safest and most effective therapy for each patient.
Features:
• Get comprehensive coverage of mechanisms, clinical implications, and limitations of current therapeutic interventions, including drugs, and catheter and surgical ablation.
• Gain a better visual understanding thanks to more than 1,100 illustrations (over 100 are new!), an increased number of 3-D color anatomical mapping images, ECG examples, photographs of equipment, and procedural diagrams.
• Stay up to date with information on new technologies of ablation and pitfalls of interpreting data; innovative new catheters; new drug information; and new tables summarizing SVT and VT criteria.
• Benefit from Dr. Josephson's decades of experience as "the father of clinical cardiac electrophysiology," and learn from his proven approaches and methods in this challenging area. Now with the print edition, enjoy the bundled interactive eBook edition, offering tablet, smartphone, or online access to:
• Complete content with enhanced navigation.
• Powerful search tools and smart navigation cross-links that pull results from content in the book, your notes, and even the web.
• Cross-linked pages, references, and more for easy navigation.
• Highlighting tool for easier reference of key content throughout the text.
• Ability to take and share notes with friends and colleagues.
• Quick reference tabbing to save your favorite content for future use.
Table of contents:
Chapter 1: Electrophysiologic Investigation: Technical Aspects
Personnel
Equipment
Electrode Catheters
Laboratory Organization
Recording and Stimulation Apparatus
Junction Box
Recording Apparatus
Stimulator
Cardioverter/Defibrillator
Figure 1-1
Figure 1-2
Figure 1-3
Figure 1-4
Figure 1-5
Figure 1-6
Figure 1-7
Figure 1-8
Figure 1-9
Figure 1-10
Figure 1-11
Figure 1-12
Figure 1-13
Figure 1-14
Figure 1-15
Figure 1-16
Figure 1-17
Cardiac Catheterization Technique
Femoral Vein Approach
Inadvertent Puncture of the Femoral Artery
Upper Extremity Approach
Right Atrium
Left Atrium
Right Ventricle
Left Ventricle
His Bundle Electrogram
Table 1-1: Catheter Approach for Electrophysiologic Study
Figure 1-18
Figure 1-19
Figure 1-20
Figure 1-21
Figure 1-22
Figure 1-23
Figure 1-24
Risks and Complications
Significant Hemorrhage
Thromboembolism
Phlebitis
Arrhythmias
Complications of Left Ventricular Studies
Tamponade
Artifacts
Figure 1-25
References
Chapter 2: Electrophysiologic Investigation: General Concepts
Chapter 2 Introduction
Electrogram Recordings
Measurement of Conduction Intervals
His Bundle Electrogram
Assessment of “H”-V Interval
Establishing Relationship of the His Bundle Deflection to other Electrograms: Role of Catheter Position
Simultaneous Left-sided and Right-sided Recordings
His Bundle Pacing
A-H Interval
H-V Interval
Intra-atrial Conduction
Intraventricular Conduction
Figure 2-1
Figure 2-2
Figure 2-3
Figure 2-4
Figure 2-5
Figure 2-6
Figure 2-7
Figure 2-8
Table 2-1: Normal Conduction Intervals in Adults
Figure 2-9
Figure 2-10
Figure 2-11
Figure 2-12
Figure 2-13
Figure 2-14
Figure 2-15
Figure 2-16
Description of Electrograms
Descriptive Characteristics
Quantitative Characteristics
Left Ventricular Endocardial Activation
Table 2-2: Summary of Electrogram Characteristics in Normal Left Ventricles
Table 2-3: Electrogram Amplitude and Duration Characteristics in Normal Left Ventricles by Left Ventricular Sitea Number
Figure 2-17
Figure 2-18
Programmed Stimulation
Incremental Pacing
Refractory Periods
Cycle Length Responsiveness of Refractory Periods
Dispersion of Refractoriness
Patterns of Response to Atrial Extrastimuli
The Atrium as a Limiting Factor in A-V Conduction
Patterns of Response to Ventricular Extrastimuli
Comparison of Antegrade and Retrograde Conduction
Figure 2-19
Figure 2-20
Figure 2-21
Figure 2-22
Figure 2-23
Figure 2-24
Figure 2-25
Figure 2-26
Figure 2-27
Figure 2-28
Table 2-4: Definition of Terms
Figure 2-29
Figure 2-30
Figure 2-31
Figure 2-32
Figure 2-33
Figure 2-34
Figure 2-35
Figure 2-36
Figure 2-37
Table 2-5: Normal Refractory Periods in Adults
Figure 2-38
Figure 2-39
Figure 2-40
Figure 2-41
Figure 2-42
Figure 2-43
Figure 2-44
Figure 2-45
Figure 2-46
Figure 2-47
Figure 2-48
Figure 2-49
Figure 2-50
Figure 2-51
Figure 2-52
Figure 2-53
Figure 2-54
Figure 2-55
Figure 2-56
Figure 2-57
Figure 2-58
Figure 2-59
Figure 2-60
Figure 2-61
References
Chapter 3: Sinus Node Function
Chapter 3 Introduction
Electrocardiographic Features of Sinus Node Dysfunction
Sinus Bradycardia
Sinoatrial Block and Sinus Arrest
Bradycardia–tachycardia Syndrome
Electrocardiographic Monitoring of Patients Suspected of Having Sinus Node Dysfunction
Assessment of Autonomic Tone
Figure 3-1
Electrophysiologic Evaluation of Sinus Node Function
Sinoatrial Conduction Time
Sinus Node Electrogram
Sinoatrial Conduction Time in Patients with Sick Sinus Syndrome
Sinus Node Recovery Time
Effect of Atropine and Autonomic Blockade on Sinus Node Recovery Time in Normal Persons
Results of Atrial Pacing in Patients Suspected of Having Sinus Node Dysfunction
Figure 3-2
Figure 3-3
Figure 3-4
Figure 3-5
Figure 3-6
Figure 3-7
Figure 3-8
Table 3-1: Normal Upper Limits for Sinoatrial Conduction Time (SACTa, msec)
Figure 3-9
Figure 3-10
Figure 3-11
Figure 3-12
Table 3-2: Diagnostic Evaluation of Sinus Node Function
Table 3-3: Normal Values (msec) for Maximum Sinus Node Recovery Time (SNRTmax) and Maximum Corrected Sinus Node Recovery Time (CSNRTmax)
Figure 3-13
Figure 3-14
Figure 3-15
Figure 3-16
Figure 3-17
Figure 3-18
Figure 3-19
Effect of Drugs on Sinus Node Recovery Time and Sinoatrial Conduction Time
Digoxin
Propranolol
Calcium Blockers
Antiarrhythmic Agents
Vagal Hypersensitivity (Neurocirculatory) Syndromes
Figure 3-20
Figure 3-21
Therapeutic Implications
References
Chapter 4: Atrioventricular Conduction
Chapter 4 Introduction
Table 4-1: Atrioventricular Block
Table 4-2: Sites of Atrioventricular Block
Atrium
Figure 4-1
Figure 4-2
Figure 4-3
Figure 4-4
Figure 4-5
Figure 4-6
Atrioventricular Node
Figure 4-7
Figure 4-8
Figure 4-9
Figure 4-10
Figure 4-11
Figure 4-12
His Bundle
Figure 4-13
Figure 4-14
Figure 4-15
Figure 4-16
Figure 4-17
Infra-His Conduction System
Figure 4-18
Figure 4-19
Figure 4-20
Figure 4-21
Figure 4-22
Figure 4-23
Table 4-3: Site of 2:1 A-V Block
Figure 4-24
Table 4-4: Significance of Retrograde Conduction in the Presence of Antegrade Block
Figure 4-25
Paroxysmal A-V Block
Figure 4-26
Value of Intracardiac Studies in the Evaluation of A-V Conduction Disturbances
Figure 4-27
Figure 4-28
Figure 4-29
Figure 4-30
Suppression of A-V Conduction by Ventricular Stimulation
Figure 4-31
Therapeutic Considerations
Table 4-5: Indications for Cardiac Pacing in Chronic Heart Block
Figure 4-32
References
Chapter 5: Intraventricular Conduction Disturbances
Chapter 5 Introduction
Figure 5-1
Figure 5-2
Figure 5-3
Definitions
Site of “Block” or Conduction Delay during Bundle Branch Block
Chronic Right Bundle Branch Block
Left Bundle Branch Block
Transient Bundle Branch Block
Figure 5-4
Figure 5-5
Figure 5-6
Figure 5-7
Figure 5-8
Figure 5-9
Figure 5-10
Figure 5-11
Figure 5-12
Figure 5-13
Figure 5-14
Figure 5-15
Table 5-1: Relationship between QRS Axis and Activation Sequence
Table 5-2: Results of Left Ventricular Mapping
Figure 5-16
Figure 5-17
Figure 5-18
Figure 5-19
Figure 5-20
Figure 5-21
Figure 5-22
Figure 5-23
Figure 5-24
Figure 5-25
Figure 5-26
Figure 5-27
Figure 5-28
Figure 5-29
Figure 5-30
Figure 5-31
Figure 5-32
Figure 5-33
Clinical Relevance of Intraventricular Conduction Disturbances
Role of Electrophysiologic Studies in Predicting Risk of Heart Block
Methods to Identify Patients at Risk of Developing A-V Block
Alternating Bundle Branch Block
Syncope and Sudden Death in Patients with Bundle Branch Block
Figure 5-34
Figure 5-35
Figure 5-36
Figure 5-37
Figure 5-38
Figure 5-39
Figure 5-40
Figure 5-41
Figure 5-42
Therapeutic Implications
Table 5-3: Intraventricular Conduction Disturbances: Assessment of HPS Reserve
Table 5-4: Intraventricular Conduction Disturbances: Recommendations for Clinical Cardiac Pacing
References
Chapter 6: Miscellaneous Phenomena Related to Atrioventricular Conduction
Chapter 6 Introduction
Concealed Conduction
Figure 6-1
Figure 6-2
Figure 6-3
Figure 6-4
Figure 6-5
Figure 6-6
Figure 6-7
Figure 6-8
Figure 6-9
Gap Phenomenon
Table 6-1: Classification of Gap Phenomena in the Human Heart
Figure 6-10
Figure 6-11
Figure 6-12
Figure 6-13
Figure 6-14
Supernormality
Figure 6-15
Figure 6-16
Figure 6-17
Figure 6-18
Figure 6-19
Figure 6-20
References
Chapter 7: Ectopic Rhythms and Premature Depolarizations
Chapter 7 Introduction
Atrial Depolarizations
Figure 7-1
Figure 7-2
Figure 7-3
Figure 7-4
Figure 7-5
Figure 7-6
Junctional (His Bundle or A-V Nodal) Depolarizations
Figure 7-7
Figure 7-8
Figure 7-9
Figure 7-10
Figure 7-11
Figure 7-12
Figure 7-13
Figure 7-14
Figure 7-15
Fascicular Depolarizations
Figure 7-16
Figure 7-17
Figure 7-18
Figure 7-19
Ventricular Depolarizations
Figure 7-20
Figure 7-21
Figure 7-22
Figure 7-23
References
Chapter 8: Supraventricular Tachycardias
Chapter 8 Introduction
Mechanisms of Supraventricular Tachycardia
Methods of Evaluation
Table 8-1: Electrophysiologic Evaluation of Supraventricular Tachycardia
Table 8-2: Mechanisms of SVT (2,789 Patients)
Supraventricular Tachycardia Resulting from Atrioventricular Nodal Reentry
Mechanisms of Initiation of Atrioventricular Nodal Reentry
Determinants for the Induction of Atrioventricular Nodal Reentry
Atrial Activation Sequence and the P–QRS Relationship During Supraventricular Tachycardia
Effect of Bundle Branch Block During Atrioventricular Nodal Reentrant Supraventricular Tachycardia
Requirement of the Atrium and Ventricle
Requirement of the Atrium in AVNRT
Responses to Stimulation During AVNRT—Role in Defining Atrial and Subnodal Participation
Role of Atrial Activation Patterns in Evaluating the Role of the Atrium
Requirement of Subnodal Structures in AVNRT
The Role of Atrial or Ventricular Pacing in Analyzing Upper and Lower Final Common Pathways
Pharmacologic and Physiologic Maneuvers
Figure 8-1
Figure 8-2
Figure 8-3
Figure 8-4
Figure 8-5
Figure 8-6
Figure 8-7
Figure 8-8
Figure 8-9
Figure 8-10
Figure 8-11
Figure 8-12
Figure 8-13
Figure 8-14
Figure 8-15
Figure 8-16
Figure 8-17
Figure 8-18
Figure 8-19
Figure 8-20
Figure 8-21
Figure 8-22
Figure 8-23
Figure 8-24
Figure 8-25
Figure 8-26
Figure 8-27
Figure 8-28
Figure 8-29
Figure 8-30
Figure 8-31
Figure 8-32
Figure 8-33
Figure 8-34
Figure 8-35
Figure 8-36
Figure 8-37
Figure 8-38
Figure 8-39
Table 8-3: AVNRT: Evidence That the Atrium is not Necessary
Figure 8-40
Figure 8-41
Figure 8-42
Figure 8-43
Figure 8-44
Figure 8-45
Figure 8-46
Figure 8-47
Figure 8-48
Figure 8-49
Figure 8-50
Figure 8-51
Figure 8-52
Figure 8-53
Figure 8-54
Figure 8-55
Figure 8-56
Figure 8-57
Figure 8-58
Figure 8-59
Figure 8-60
Figure 8-61
Figure 8-62
Figure 8-63
Figure 8-64
Figure 8-65
Figure 8-66
Figure 8-67
Figure 8-68
Figure 8-69
Figure 8-70
Figure 8-71
Figure 8-72
Table 8-4: AVNRT: Evidence That the His Bundle is not Involved
Figure 8-73
Figure 8-74
Figure 8-75
Figure 8-76
Figure 8-77
Figure 8-78
Table 8-5: Criteria for A-V Nodal Reentrant SVT
Supraventricular Tachycardia Resulting from Concealed Atrioventricular Bypass Tracts
Mechanism of Initiation
Atrial Activation Sequence and P–QRS Relationship During Supraventricular Tachycardia
Effect of Bundle Branch Block During Supraventricular Tachycardia Using Concealed Bypass Tracts
Requirement of Atrium and Ventricle During Supraventricular Tachycardia Using Concealed Bypass Tracts
Electrical Stimulation During Supraventricular Tachycardia Using a Concealed Bypass Tract
Effects of Pharmacologic and Physiologic Maneuvers During Supraventricular Tachycardia
Figure 8-79
Figure 8-80
Figure 8-81
Figure 8-82
Figure 8-83
Figure 8-84
Figure 8-85
Figure 8-86
Figure 8-87
Figure 8-88
Figure 8-89
Figure 8-90
Figure 8-91
Figure 8-92
Figure 8-93
Figure 8-94
Figure 8-95
Figure 8-96
Figure 8-97
Figure 8-98
Figure 8-99
Figure 8-100
Figure 8-101
Figure 8-102
Figure 8-103
Figure 8-104
Figure 8-105
Table 8-6: Location of Concealed Bypass Tracts (1,349 Patients)
Figure 8-106
Figure 8-107
Figure 8-108
Figure 8-109
Figure 8-110
Figure 8-111
Figure 8-112
Figure 8-113
Figure 8-114
Figure 8-115
Figure 8-116
Figure 8-117
Figure 8-118
Figure 8-119
Figure 8-120
Figure 8-121
Figure 8-122
Figure 8-123
Figure 8-124
Figure 8-125
Figure 8-126
Figure 8-127
Figure 8-128
Table 8-7: Criteria for A-V Reentrant SVT
Figure 8-129
Figure 8-130
Figure 8-131
Figure 8-132
Figure 8-133
Figure 8-134
Figure 8-135
Figure 8-136
Figure 8-137
Figure 8-138
Supraventricular Tachycardia Resulting from Intra-Atrial or Sinus Node Reentry
Mechanism of Initiation of Supraventricular Tachycardias Resulting from Intra-atrial Reentry
Atrial Activation Sequence in P–QRS Relationship During Supraventricular Tachycardias with Intra-atrial Reentry
Requirement of the Atrium and Ventricles and Influence of Bundle Branch Block on Supraventricular Tachycardia Resulting from Intra-atrial Reentry
Effects of Stimulation During Supraventricular Tachycardias Resulting from Intra-atrial Reentry
Physiologic and Pharmacologic Maneuvers During Supraventricular Tachycardias Resulting from Intra-atrial Reentry
Table 8-8: Criteria for “Sinus Node” Reentrant SVT
Table 8-9: Criteria for Intra-atrial Reentrant SVT (Distant from the Sinus Node)
Figure 8-139
Figure 8-140
Figure 8-141
Figure 8-142
Figure 8-143
Figure 8-144
Figure 8-145
Figure 8-146
Figure 8-147
Figure 8-148
Automatic Atrial Tachycardia
Figure 8-149
Figure 8-150
Figure 8-151
Figure 8-152
Figure 8-153
Figure 8-154
Figure 8-155
Table 8-10: Criteria for Automatic Atrial Tachycardia
Atrial Tachycardia due to Triggered Activity
Figure 8-156
Figure 8-157
Figure 8-158
Figure 8-159
Distinguishing Atrial Tachycardia from AVNRT and AVRT
Figure 8-160
Figure 8-161
Table 8-11: Methods to Distinguish AVNRT versus AVRT
Multiple SVT Mechanisms in Individual Patients
Figure 8-162
Figure 8-163
Figure 8-164
Figure 8-165
Figure 8-166
Figure 8-167
Overview
Table 8-12: Criteria for Atrial Tachycardia Resulting from Triggered Activity
Figure 8-168
Figure 8-169
Figure 8-170
References
Chapter 9: Atrial Flutter and Fibrillation
Chapter 9 Introduction
Electrophysiologic and Anatomic Substrates of Macroreentrant Atrial Tachycardia (Typical and Atypical Atrial Flutter) and Fibrillation
Conduction Defects in Patients with Atrial Fibrillation and Flutter
Atrial Refractoriness
Atrial Vulnerability
Figure 9-1
Figure 9-2
Figure 9-3
Figure 9-4
Figure 9-5
Figure 9-6
Figure 9-7
Figure 9-8
Figure 9-9
Figure 9-10
Table 9-1: Intra-atrial Conduction Velocity in Patients With and Without Atrial Fibrillation
Figure 9-11
Figure 9-12
Figure 9-13
Figure 9-14
Figure 9-15
Table 9-2: Effect of Stimulation Frequency on Effective Refractory Period
Figure 9-16
Atrial Flutter
Induction of Atrial Flutter
Activation Mapping and Programmed Stimulation During Flutter
Characterization of the Reentrant Circuit in Atrial Flutter
Termination of Atrial Flutter
Effects of Pharmacologic Agents on Reentrant Atrial Flutter
Atrioventricular Conduction During Flutter
Figure 9-17
Figure 9-18
Figure 9-19
Figure 9-20
Figure 9-21
Figure 9-22
Figure 9-23
Figure 9-24
Figure 9-25
Figure 9-26
Figure 9-27
Figure 9-28
Figure 9-29
Figure 9-30
Figure 9-31
Figure 9-32
Figure 9-33
Figure 9-34
Figure 9-35
Figure 9-36
Figure 9-37
Figure 9-38
Figure 9-39
Figure 9-40
Figure 9-41
Figure 9-42
Figure 9-43
Figure 9-44
Figure 9-45
Figure 9-46
Atrial Fibrillation
Mapping During Atrial Fibrillation
Stimulation During Atrial Fibrillation
Relationship Between Atrial Flutter and Fibrillation
Miscellaneous Uses of Electrophysiology Studies
Figure 9-47
Figure 9-48
Figure 9-49
Figure 9-50
Figure 9-51
Figure 9-52
Figure 9-53
Figure 9-54
Table 9-3: Source of Atrial Activation Wavefronts on the Right Atrial Free Wall in Patients with Chronic and Nonsustained Atrial Fibrillation (mean)
Figure 9-55
Figure 9-56
Table 9-4: Fibrillation Intervals in Patients with Chronic and Nonsustained Atrial Fibrillation (ms; mean + SD)
Table 9-5: Incidence and Length of Conduction Block in Patients with Chronic and Nonsustained Atrial Fibrillation (mean ± SD)
Figure 9-57
Figure 9-58
Figure 9-59
Figure 9-60
Figure 9-61
Figure 9-62
Figure 9-63
Figure 9-64
Summary
References
Chapter 10: Preexcitation Syndromes
Chapter 10 Introduction
Figure 10-1
Atrioventricular Bypass Tracts
Figure 10-2
Electrophysiologic Properties of A-V Bypass Tracts
Figure 10-3
Figure 10-4
Figure 10-5
Electrophysiologic Evaluation in Patients with Wolff–Parkinson–White Syndrome
Diagnosis of an A-V Bypass Tract
Figure 10-6
Figure 10-7
Figure 10-8
Figure 10-9
Figure 10-10
Figure 10-11
Figure 10-12
Mode of Initiation of Tachycardias
Orthodromic Tachycardia
Figure 10-13
Figure 10-14
Figure 10-15
Figure 10-16
Figure 10-17
Figure 10-18
Figure 10-19
Figure 10-20
Figure 10-21
Figure 10-22
Figure 10-23
Figure 10-24
Figure 10-25
Preexcited Tachycardias
Table 10-1: Mechanisms of Regular Preexcited Tachycardias
Figure 10-26
Table 10-2: Electrophysiologic Substrate of Antidromic Tachycardia
Table 10-3: Characteristics of A-V Nodal Reentry with an Innocent Bystander Atrioventricular Bypass Tract
Figure 10-27
Figure 10-28
Figure 10-29
Figure 10-30
Figure 10-31
Figure 10-32
Figure 10-33
Figure 10-34
Figure 10-35
Figure 10-36
Figure 10-37
Figure 10-38
Table 10-4: Methods to Exclude A-V Nodal Reentry as a Cause of a Preexcited Tachycardia
Figure 10-39
Figure 10-40
Table 10-5: Rate Changes During Preexcited Tachycardias Due to Changes in Retrograde Conduction
Table 10-6: Rate Changes During Regular Preexcited Tachycardias Due to Changes in Anterograde Conduction
Table 10-7: Effect of Carotid Sinus Pressure on Preexcited Tachycardias
Figure 10-41
Figure 10-42
Figure 10-43
Atrial Fibrillation
Figure 10-44
Localization of the Bypass Tract
Figure 10-45
Relation of Local Ventricular Electrograms to Delta Wave
Figure 10-46
Figure 10-47
Figure 10-48
Figure 10-49
Pacing from Multiple Atrial Sites
Figure 10-50
Retrograde Atrial Activation
Figure 10-51
Figure 10-52
Figure 10-53
Figure 10-54
Figure 10-55
Figure 10-56
Figure 10-57
Figure 10-58
Figure 10-59
Table 10-8: Methods of Distinguishing a Bypass Tract from Normal Retrograde Conduction Due to A-V Nodal Reentry
Figure 10-60
Figure 10-61
Effect of Bundle Branch Block during Orthodromic Tachycardia
Figure 10-62
Figure 10-63
Figure 10-64
Figure 10-65
Figure 10-66
Figure 10-67
Figure 10-68
Figure 10-69
Figure 10-70
Direct Recording of Bypass Tract Potentials
Figure 10-71
Figure 10-72
Figure 10-73
Figure 10-74
Figure 10-75
Figure 10-76
Figure 10-77
Figure 10-78
Role of the Bypass Tract in Genesis of Arrhythmias
Figure 10-79
Figure 10-80
Figure 10-81
Figure 10-82
Figure 10-83
Figure 10-84
Figure 10-85
Figure 10-86
Figure 10-87
Determination of the Antegrade Refractory Period of the Bypass Tract
Intermittent Preexcitation
Figure 10-88
Effect of Antiarrhythmic Agents on Preexcitation
Figure 10-89
Exercise Testing in Wolff–Parkinson–White Syndrome
Determination of the Antegrade Refractory Period of the Bypass Tract by Programmed Stimulation
Figure 10-90
Figure 10-91
Termination of Orthodromic Tachycardia
Multiple Bypass Tracts
Figure 10-92
Figure 10-93
Table 10-9: Evidence of Multiple Bypass Tracts
Figure 10-94
Figure 10-95
Figure 10-96
Figure 10-97
Figure 10-98
Figure 10-99
Figure 10-100
Figure 10-101
Figure 10-102
Figure 10-103
Atrioventricular Nodal “Bypass Tracts”—The Lown–Ganong–Levine Syndrome
Electrophysiologic Properties
Atrial Pacing
Response to Atrial Premature Depolarizations
Ventriculoatrial Conduction
Response to Pharmacologic and Physiologic Maneuvers
Role of the Bypass Tract in Arrhythmias
Therapeutic Implications
Figure 10-104
Figure 10-105
Figure 10-106
Figure 10-107
Figure 10-108
Figure 10-109
Figure 10-110
Figure 10-111
Accessory Pathways with Anterograde Decremental Conduction and Fasciculoventricular Pathways
Slowly Conducting Accessory Pathways
Electrophysiologic Manifestations
Atriofascicular and Long Atrioventricular Bypass Tracts
Short Slowly Conducting Atrioventricular Bypass Tracts
Nodofascicular and Nodoventricular Bypass Tracts
Tachycardias Associated with Atriofascicular, Slowly Conducting A-V, Nodofascicular, and Nodoventricular Bypass Tracts
Therapeutic Implications
Fasciculoventricular Bypass Tracts
Table 10-10: Tachycardias Using Decremental (“Mahaim”) Fibers
Figure 10-112
Figure 10-113
Figure 10-114
Figure 10-115
Figure 10-116
Figure 10-117
Figure 10-118
Figure 10-119
Figure 10-120
Figure 10-121
Figure 10-122
Figure 10-123
Figure 10-124
Figure 10-125
Figure 10-126
Figure 10-127
Figure 10-128
Figure 10-129
Figure 10-130
Figure 10-131
Figure 10-132
Figure 10-133
Figure 10-134
Figure 10-135
Table 10-11: Tachycardia Types
Table 10-12: Electrophysiologic Features of Tachycardias Associated with Atrio/Nodofascicular Bypass Tracts and Atrio/Nodoventricular Bypass Tracts
Figure 10-136
Figure 10-137
Figure 10-138
Figure 10-139
Figure 10-140
Figure 10-141
Figure 10-142
Figure 10-143
Figure 10-144
Figure 10-145
Figure 10-146
Figure 10-147
References
Chapter 11: Recurrent Ventricular Tachycardia
Chapter 11 Introduction
Definitions of Ventricular Tachycardias
Morphology
Duration
Classification of Ventricular Tachycardia QRS Complexes
Diagnosis of Ventricular Tachycardia
Use of His Bundle Recordings in Diagnosing Ventricular Tachycardia
Limitations of His Bundle Recordings for the Diagnosis of Ventricular Tachycardia
Table 11-1: Characteristics of Wide Complex Tachycardia Favoring Diagnosis of VT
Figure 11-1
Figure 11-2
Figure 11-3
Figure 11-4
Figure 11-5
Figure 11-6
Figure 11-7
Figure 11-8
Figure 11-9
Figure 11-10
Figure 11-11
Figure 11-12
Pathophysiologic Substrate for Ventricular Tachyarrhythmias
Anatomic Substrate
Electrophysiologic Substrate
Figure 11-13
Figure 11-14
Figure 11-15
Figure 11-16
Figure 11-17
Figure 11-18
Table 11-2: Influence of Coronary Artery Disease and Clinical Arrhythmia
Figure 11-19
Figure 11-20
Figure 11-21
Figure 11-22
Figure 11-23
Figure 11-24
Figure 11-25
Table 11-3: Influence of Underlying Heart Disease
Figure 11-26
Table 11-4: Influence of Cardiomyopathy and Clinical Arrhythmia
Figure 11-27
Figure 11-28
Figure 11-29
Figure 11-30
Figure 11-31
Figure 11-32
Figure 11-33
Figure 11-34
Figure 11-35
Figure 11-36
Figure 11-37
Figure 11-38
Table 11-5: Definitions
Figure 11-39
Figure 11-40
Figure 11-41
Table 11-6: Data Based on Whole Left Ventricle Analysis
Table 11-7: Data Based on Adjacent Left Ventricle Sites
Mechanisms of Ventricular Tachycardia
Initiation of Ventricular Tachycardias
Protocol of Programmed Stimulation
Number of Extrastimuli
Influence of Drive Cycle Length
Multiple Sites of Stimulation
Role of Increasing Current
Initiation of Sustained Uniform Ventricular Tachycardia
Relationship of Coupling Intervals and Cycle Length Initiating Tachycardia to the Onset of Ventricular Tachycardia and the Initial Ventricular Tachycardia Cycle Length
Relationship of Stimulation Site to Initiation of Ventricular Tachycardia
Relationship of Initiation of VT to Conduction Delay, Block, and/or Continuous Activity
Role of the His–Purkinje System in Initiating and Maintaining Sustained Uniform Ventricular Tachycardia
Initiation of Polymorphic Ventricular Tachycardia-Ventricular Fibrillation
Initiation of Polymorphic Ventricular Tachycardia-Ventricular Fibrillation with Normal QT Intervals
Initiation of Polymorphic Ventricular Tachycardia-Ventricular Fibrillation in the Presence of Long QT Intervals
Mechanism of QRS Morphology in Polymorphic Ventricular Tachycardia
Initiation of Monomorphic Nonsustained Ventricular Tachycardia
Response of Sustained Uniform Ventricular Tachycardia to Stimulation
Protocol for Stimulation During Sustained VT
Manifest and Concealed Perpetuation of Ventricular Tachycardia
Resetting of Ventricular Tachycardia
Return Cycle Versus Ventricular Tachycardia Cycle Length
Resetting with Fusion
Site Specificity of Resetting
Resetting Response Curves
Response of Ventricular Tachycardia to Overdrive Pacing—Continuous Resetting (Entrainment)
Termination of Sustained Ventricular Tachycardia
Mechanism of Termination and Relationship to Resetting Phenomena
Modification of Factors Influencing Termination of Ventricular Tachycardia
Figure 11-42
Figure 11-43
Figure 11-44
Figure 11-45
Figure 11-46
Figure 11-47
Table 11-8: Relationship of Induction Mode to RVERPa
Figure 11-48
Table 11-9: Drive Cycle Lengths of Programmed Stimulation Required for VT Induction
Figure 11-49
Figure 11-50
Table 11-10: Site of Stimulation Required for Arrhythmia Conduction
Table 11-11: Protocol of Programmed Stimulation for VT
Figure 11-51
Figure 11-52
Figure 11-53
Figure 11-54
Figure 11-55
Figure 11-56
Figure 11-57
Figure 11-58
Figure 11-59
Table 11-12: Mode of Initiation of VT Compared with Cycle Length of the 126 Morphologically Distinct Tachycardias in 104 Patients
Figure 11-60
Figure 11-61
Figure 11-62
Figure 11-63
Figure 11-64
Figure 11-65
Figure 11-66
Figure 11-67
Figure 11-68
Figure 11-69
Figure 11-70
Figure 11-71
Figure 11-72
Figure 11-73
Figure 11-74
Figure 11-75
Figure 11-76
Figure 11-77
Figure 11-78
Figure 11-79
Figure 11-80
Figure 11-81
Figure 11-82
Figure 11-83
Figure 11-84
Figure 11-85
Figure 11-86
Figure 11-87
Figure 11-88
Figure 11-89
Figure 11-90
Figure 11-91
Figure 11-92
Figure 11-93
Figure 11-94
Figure 11-95
Figure 11-96
Figure 11-97
Figure 11-98
Figure 11-99
Figure 11-100
Figure 11-101
Figure 11-102
Figure 11-103
Figure 11-104
Table 11-13: Relationship of Mode of Termination of VT with VT Cycle Length (139 Patients)
Figure 11-105
Figure 11-106
Figure 11-107
Figure 11-108
Figure 11-109
Figure 11-110
Figure 11-111
Figure 11-112
Figure 11-113
Figure 11-114
Figure 11-115
Figure 11-116
Figure 11-117
Figure 11-118
Figure 11-119
Figure 11-120
Figure 11-121
Figure 11-122
Figure 11-123
Figure 11-124
Figure 11-125
Figure 11-126
Figure 11-127
Figure 11-128
Figure 11-129
Figure 11-130
Figure 11-131
Figure 11-132
Figure 11-133
Figure 11-134
Figure 11-135
Figure 11-136
Figure 11-137
Figure 11-138
Figure 11-139
Figure 11-140
Figure 11-141
Figure 11-142
Figure 11-143
Figure 11-144
Figure 11-145
Figure 11-146
Figure 11-147
Figure 11-148
Figure 11-149
Figure 11-150
Figure 11-151
Figure 11-152
Figure 11-153
Figure 11-154
Figure 11-155
Figure 11-156
Figure 11-157
Figure 11-158
Figure 11-159
Figure 11-160
Figure 11-161
Figure 11-162
Figure 11-163
Figure 11-164
Figure 11-165
Figure 11-166
Figure 11-167
Figure 11-168
Figure 11-169
Figure 11-170
Figure 11-171
Figure 11-172
Figure 11-173
Figure 11-174
Figure 11-175
Figure 11-176
Figure 11-177
Figure 11-178
Figure 11-179
Figure 11-180
Figure 11-181
Figure 11-182
Figure 11-183
Table 11-14: Effects of Programmed Ventricular Stimulation (PVS) During VT (123 Patients)
Figure 11-184
Figure 11-185
Figure 11-186
Figure 11-187
Figure 11-188
Figure 11-189
Figure 11-190
Figure 11-191
Figure 11-192
Figure 11-193
Figure 11-194
Figure 11-195
Figure 11-196
Figure 11-197
Figure 11-198
Figure 11-199
Figure 11-200
Figure 11-201
Figure 11-202
Figure 11-203
Figure 11-204
Table 11-15: Effect of Procainamide on Mode of Termination of VT (23 Patients)
Figure 11-205
Figure 11-206
Figure 11-207
Effect of Drugs on Ventricular Tachycardia
Figure 11-208
Figure 11-209
Figure 11-210
Figure 11-211
Figure 11-212
Localization of the Site of Origin of Ventricular Tachycardia
General Methods of Catheter Mapping
Acquisition and Interpretation of Activation Mapping During Ventricular Tachycardia
Continuous Activity and Diastolic Bridging
Role of Programmed Stimulation in Identifying the Critical Sites in a Reentrant Circuit
Relationship of Mapping Data to Heart Disease
Multiple Ventricular Tachycardia Morphologies: Relationship to Site of Origin
Validation of Catheter Mapping
Sinus Rhythm Mapping
Relationship of QRS Morphologies to Sites of Origin of Tachycardias
Relationship of QRS Patterns of Sites of Origin of Ventricular Tachycardia
Role of Pace Mapping in Determining the Site of Origin of Ventricular Tachycardia
Figure 11-213
Figure 11-214
Figure 11-215
Figure 11-216
Figure 11-217
Figure 11-218
Figure 11-219
Figure 11-220
Figure 11-221
Figure 11-222
Figure 11-223
Figure 11-224
Figure 11-225
Figure 11-226
Figure 11-227
Figure 11-228
Figure 11-229
Figure 11-230
Figure 11-231
Figure 11-232
Figure 11-233
Figure 11-234
Figure 11-235
Figure 11-236
Figure 11-237
Figure 11-238
Figure 11-239
Figure 11-240
Figure 11-241
Figure 11-242
Figure 11-243
Figure 11-244
Figure 11-245
Figure 11-246
Figure 11-247
Figure 11-248
Figure 11-249
Figure 11-250
Figure 11-251
Figure 11-252
Figure 11-253
Figure 11-254
Figure 11-255
Figure 11-256
Figure 11-257
Figure 11-258
Figure 11-259
Figure 11-260
Figure 11-261
Figure 11-262
Figure 11-263
Figure 11-264
Figure 11-265
Figure 11-266
Figure 11-267
Figure 11-268
Figure 11-269
Figure 11-270
Figure 11-271
Figure 11-272
Figure 11-273
Figure 11-274
Figure 11-275
Ventricular Stimulation in Miscellaneous Disorders
References
Chapter 12: Evaluation of Antiarrhythmic Agents
Chapter 12 Introduction
Classification of Antiarrhythmic Agents
In Vitro Classification of Antiarrhythmic Agents
Table 12-1: In Vitro Electrophysiologic Characteristics
Evaluation of Electrophysiologic Effects of Drugs in Humans
Table 12-2: In Vivo Electrophysiologic Characteristics of Antiarrhythmic Drugs Electrophysiologic Effects
Table 12-3: In Vivo Electrophysiologic Characteristics of Antiarrhythmic Drugs Electrocardiographic Effects
Table 12-4: Effects of Lidocaine on QRS Width and Electrogram Duration (msec)
Figure 12-1
Figure 12-2
Figure 12-3
Table 12-5: Effects of Procainamide on QRS Width and Electrogram Duration (msec)
Table 12-6: Amplitude (in mV)
Figure 12-4
Figure 12-5
Figure 12-6
Figure 12-7
Figure 12-8
Table 12-7: Limitations in Evaluating Antiarrhythmic Drug Action
Selection of Antiarrhythmic Drug Therapy for Reentrant Tachycardias
Use of Programmed Stimulation
Study Protocol
Selection of Pharmacologic Therapy for Ventricular Tachyarrhythmias
Factors Predicting Outcome of Electrophysiologic Study
Effect of Drugs on VT Cycle Length
Empiric Use of Antiarrhythmic Drugs in the Setting of Concurrent ICD Therapy
Predictability of Drug Effect on Tachycardia Cycle Length
Analysis of Drug Effects on Tachycardia Cycle Length Using Resetting and Entrainment
Mechanism of Drug Effect on Inducibility and Termination of VT
Selection of Antiarrhythmic Agents for Supraventricular Tachyarrhythmias
Proarrhythmic Effects of Antiarrhythmic Agents
Electrophysiologic Studies and Proarrhythmia
Figure 12-9
Table 12-8: Dosage and Therapeutic Serum Concentrations for Antiarrhythmic Agents
Figure 12-10
Figure 12-11
Figure 12-12
Figure 12-13
Figure 12-14
Figure 12-15
Figure 12-16
Figure 12-17
Figure 12-18
Figure 12-19
Figure 12-20
Figure 12-21
Figure 12-22
Figure 12-23
Figure 12-24
Figure 12-25
Figure 12-26
Figure 12-27
Figure 12-28
Figure 12-29
Figure 12-30
Figure 12-31
Figure 12-32
Figure 12-33
Figure 12-34
Figure 12-35
Figure 12-36
Figure 12-37
Figure 12-38
Figure 12-39
Figure 12-40
Figure 12-41
Figure 12-42
Figure 12-43
Figure 12-44
Figure 12-45
Figure 12-46
Figure 12-47
Table 12-9: Effect of Drugs on VT Cycle Length
Figure 12-48
Figure 12-49
Table 12-10: Correlation Between Efficacy and Magnitude of Change in QTC and ERP by Type lA Drugs
Figure 12-50
Figure 12-51
Figure 12-52
Figure 12-53
Figure 12-54
Figure 12-55
Figure 12-56
Figure 12-57
Figure 12-58
References
Chapter 13: Catheter and Surgical Ablation in the Therapy of Arrhythmias
Chapter 13 Introduction
Biophysics of Current Ablation Techniques
DC Ablation
Radiofrequency Energy
Laser Ablation
Cryoablation
Ultrasound
Figure 13-1
Figure 13-2
Figure 13-3
Figure 13-4
Figure 13-5
Figure 13-6
Control of Supraventricular Arrhythmias by Ablative Techniques
Ablation of Atrioventricular Bypass Tracts and Variants of Pre-excitation
Localization of Bypass Tracts
Catheter Ablation of Bypass Tracts
Ablation of Pre-excitation Variants
Complications of Procedures
Intraoperative Mapping and Surgical Ablation of Accessory Pathways
Ablation of the A-V Junction for Ventricular Rate Control during Atrial Tachyarrhythmias
Catheter Ablation Techniques to Treat A-V Nodal Tachycardia
Junctional Tachycardia
Surgical and Catheter Ablation Techniques to Manage other Atrial Arrhythmias
Ablation of Atrial Tachycardia
Ablation of Atrial Flutter and other Macroreentrant Atrial Arrhythmias
Ablative Techniques (Surgical and Catheter-Based) for Treatment of Atrial Fibrillation
Role of Linear Lesions to the Mitral Annulus and/or Across the Roof or Posterior Left Atrial Wall
Ablation/Modification of the Autonomic Nervous System for Control of AF
Stepwise Ablation (Ablation to Termination of Persistent AF)
Focal Impulse and Rotor Modulation for Ablation of Atrial Fibrillation
Role of Ablation in “Mother Flutter”–Induced Atrial Fibrillation
Role of Ablation in “Multiple Wavelet” Atrial Fibrillation
Figure 13-7
Figure 13-8
Figure 13-9
Figure 13-10
Figure 13-11
Figure 13-12
Figure 13-13
Figure 13-14
Figure 13-15
Figure 13-16
Figure 13-17
Figure 13-18
Figure 13-19
Figure 13-20
Figure 13-21
Figure 13-22
Figure 13-23
Figure 13-24
Figure 13-25
Figure 13-26
Figure 13-27
Figure 13-28
Figure 13-29
Figure 13-30
Figure 13-31
Figure 13-32
Figure 13-33
Figure 13-34
Figure 13-35
Figure 13-36
Figure 13-37
Figure 13-38
Figure 13-39
Figure 13-40
Figure 13-41
Figure 13-42
Figure 13-43
Figure 13-44
Figure 13-45
Figure 13-46
Figure 13-47
Figure 13-48
Figure 13-49
Figure 13-50
Figure 13-51
Figure 13-52
Figure 13-53
Figure 13-54
Figure 13-55
Figure 13-56
Figure 13-57
Figure 13-58
Figure 13-59
Figure 13-60
Figure 13-61
Figure 13-62
Figure 13-63
Figure 13-64
Figure 13-65
Figure 13-66
Figure 13-67
Figure 13-68
Figure 13-69
Figure 13-70
Figure 13-71
Figure 13-72
Figure 13-73
Figure 13-74
Figure 13-75
Figure 13-76
Figure 13-77
Figure 13-78
Figure 13-79
Figure 13-80
Figure 13-81
Figure 13-82
Figure 13-83
Figure 13-84
Figure 13-85
Figure 13-86
Figure 13-87
Figure 13-88
Figure 13-89
Figure 13-90
Figure 13-91
Figure 13-92
Figure 13-93
Figure 13-94
Figure 13-95
Figure 13-96
Figure 13-97
Figure 13-98
Figure 13-99
Figure 13-100
Figure 13-101
Figure 13-102
Figure 13-103
Figure 13-104
Figure 13-105
Figure 13-106
Figure 13-107
Figure 13-108
Figure 13-109
Figure 13-110
Figure 13-111
Figure 13-112
Figure 13-113
Figure 13-114
Figure 13-115
Control of Ventricular Arrhythmias by Ablative Techniques
Role of the ECG in Localization of Tachycardias
Mapping Techniques for Ventricular Tachycardia
Activation Mapping
Pace Mapping
Entrainment Mapping
Substrate Mapping
Additional Procedures after Failed Catheter Ablation
Catheter Ablation of Ventricular Tachycardia Associated with Coronary Artery Disease
Ablation of Unstable Ventricular Tachycardias
Role of Catheter Ablation in the Treatment of Ventricular Tachycardia Associated with Nonischemic Left Ventricular Tachycardia
Role of Catheter Ablation in the Treatment of Ventricular Tachycardia Associated with Right Ventricular Dysplasia
The Role of Catheter Mapping and Ablation of Idiopathic Ventricular Tachycardias
Catheter Ablation of Miscellaneous Ventricular Tachycardias
Intraoperative Mapping and Surgical Ablation of Ventricular Tachycardias
Intraoperative Mapping Techniques for Ventricular Tachycardia
Surgical Treatment for Ventricular Tachyarrhythmias
Results of Surgery
Ventricular Tachycardia Unassociated with Coronary Artery Disease
Figure 13-116
Figure 13-117
Figure 13-118
Figure 13-119
Figure 13-120
Figure 13-121
Figure 13-122
Figure 13-123
Figure 13-124
Figure 13-125
Figure 13-126
Figure 13-127
Figure 13-128
Figure 13-129
Figure 13-130
Figure 13-131
Figure 13-132
Figure 13-133
Figure 13-134
Figure 13-135
Table 13-1: Methods of Substrate-Based Ablation for Ventricular Tachycardia
Figure 13-136
Figure 13-137
Figure 13-138
Figure 13-139
Figure 13-140
Figure 13-141
Figure 13-142
Figure 13-143
Figure 13-144
Figure 13-145
Figure 13-146
Figure 13-147
Figure 13-148
Figure 13-149
Figure 13-150
Figure 13-151
Figure 13-152
Figure 13-153
Figure 13-154
Figure 13-155
Figure 13-156
Figure 13-157
Figure 13-158
Figure 13-159
Figure 13-160
Figure 13-161
Figure 13-162
Figure 13-163
Figure 13-164
Figure 13-165
Figure 13-166
Figure 13-167
Figure 13-168
Figure 13-169
Figure 13-170
Figure 13-171
Figure 13-172
Figure 13-173
Figure 13-174
Figure 13-175
Figure 13-176
Figure 13-177
Figure 13-178
Figure 13-179
Figure 13-180
Figure 13-181
Figure 13-182
Figure 13-183
Figure 13-184
Figure 13-185
Figure 13-186
Figure 13-187
Figure 13-188
Figure 13-189
Figure 13-190
Figure 13-191
Figure 13-192
Figure 13-193
Figure 13-194
Figure 13-195
Figure 13-196
Figure 13-197
Figure 13-198
Figure 13-199
Figure 13-200
Figure 13-201
Figure 13-202
Figure 13-203
Figure 13-204
Figure 13-205
Figure 13-206
Figure 13-207
Figure 13-208
Figure 13-209
Figure 13-210
Figure 13-211
Figure 13-212
Table 13-2: Distribution of Electrogram Type Pre- and Postsubendocardial Resection
Table 13-3: Effect of Experience on Outcome: Primary Success, Clinical Success, and Noninducibility
Table 13-4: Inducibility of VT Postsubendocardial Resection and Clinical Outcome
Table 13-5: Inducibility of VT on Drugs Postsubendo-cardial Resection and Clinical Outcome
Figure 13-213
Table 13-6: Operative Mortality (SER) in Patients with Ventricular Tachycardia Early (<2 Mo) After Infarction
Table 13-7: Outcome of Surgery in Patients with VT Early (<2 Mo) or Later (>2 Mo) After Infarction
Table 13-8: Clinical Outcome of Surgery (SER) for VT Early (<2 Mo) and Late (>2 Mo) After Infarction
Figure 13-214
Figure 13-215
Conclusion
References
Appendix
Remarks
ebookshop
An aparitie | October 2, 2015 |
Autor | Mark E. Josephson MD |
Dimensiuni | 1.2 x 9 x 11 inches |
Editura | LWW |
Format | Hardback |
ISBN | 9781451187410 |
Limba | Engleza |
Nr pag | 890 |
-
1,28600 lei 1,16500 lei
Clientii ebookshop.ro nu au adaugat inca opinii pentru acest produs. Fii primul care adauga o parere, folosind formularul de mai jos.